The Kluver-Bucy syndrome (KBS) is a rare cerebral neurological disorder, characterized
by the symptoms of increased appetite, hypersexuality, hypermetamorphosis (strong
tendency to react to visual stimulus), memory disorders, visual agnosia, dullness,
aphasia, bulimia, polyuria, and polydipsia. It was first described by Kluver and Bucy
in 1939. The disorder is a neuro-behavior syndrome that occurs due to the extirpation
of a large section of the rhinencephalon and both temporal lobes in Rhesus monkeys.
In 1955, Terzian and Ore reported the first case of KBS that developed after bilateral
temporal lobectomy, which was performed for epilepsy, in a human being (
1
). The syndrome may be seen during the course of miscellaneous cerebrovascular events
such as cerebral trauma and hypoxia, mesial temporal lobe dysfunction or destruction,
and during the course of various neurological diseases such as temporal lobe epilepsy
and encephalitis (
2
,
3
). The syndrome can occur in two forms, complete and incomplete forms. Herein, we
report an 8-year-old boy diagnosed with incomplete KBS during acute encephalitis.To read this article in full you will need to make a payment
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References
- Syndrome of Kluver and Bucy; reproduced in man by bilateral removal of the temporal lobes.Neurology. 1955; 5 (cited in ref. 2): 373-380
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- Kluver-Bucy syndrome following heat stroke in a 12-year-old girl.Pediatr Neurol. 1995; 13: 73-76
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- Post-encephalitic Kluver-Bucy syndrome.Indian J Pediatr. 2000; 67: 74-76
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- Successful treatment of emotional disturbances following non-herpetic limbic encephalitis by serotonergic agents.Rinsho Shinkeigaku. 2001; 41: 805-808
Article info
Publication history
Published online: March 17, 2008
Identification
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© 2008 Elsevier Inc. Published by Elsevier Inc. All rights reserved.
