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Letter to the Editor| Volume 41, ISSUE 6, P677-678, December 2011

Causality and Emergency Medicine?

Published:February 01, 2010DOI:https://doi.org/10.1016/j.jemermed.2009.10.029
      The main goal of emergency medicine is to recognize and treat acute medical conditions and then to make an appropriate disposition (admit to a specific hospital unit or discharge home). The process for making a diagnosis and selecting a therapeutic option is well known by emergency physicians. However, the etiology of a given disorder is usually considered less important by emergency physicians, who believe that such considerations can wait and be performed in the appropriate unit (if the patient is hospitalized) or in a primary care setting. A good illustration of this is the significantly lower proportion of literature citations in PubMed of the term “causality” in the emergency medicine literature compared to some other medical specialties (Table 1). However, considering causality in the diagnostic process, as well as severity of distress, may improve the accuracy of the diagnosis (by significantly changing the post-test probability) or the choice of therapy. For instance, in the case of a patient complaining of acute chest pain, the evaluation of a possible acute coronary syndrome should include an assessment for evidence of atherosclerosis and its risk factors. The presence (or absence) of certain risk factors significantly modifies the probability of acute coronary syndrome (
      • Swap C.J.
      • Nagurney J.T.
      Value and limitations of chest pain history in the evaluation of patients with suspected acute coronary syndromes.
      ). Early treatment for acute coronary syndrome includes atherosclerosis management as well. There are other examples for which risk factors are clearly identified and the diagnosis is not simple (e.g., deep venous thrombosis risk factors in the diagnosis of pulmonary embolism, or specific allergen exposure and asthma) (
      • Chagnon I.
      • Bounameaux H.
      • Aujesky D.
      • et al.
      Comparison of two clinical prediction rules and implicit assessment among patients with suspected pulmonary embolism.
      ,
      • Sykes A.
      • Johnston S.L.
      Etiology of asthma exacerbations.
      ). The search for risk factors in the early phase of care can improve the diagnosis and assist in selecting appropriate treatments. Further studies are needed to assess how causality can improve diagnostic accuracy in emergency care.
      Table 1Proportion of Citations in PubMed that Include “Causality,” by Medical Specialty, in the Last 10 Years (Using Medical Subject Heading Terms)
      Number of Citations in the Last 10 Years, for the Specialty Number of Citations in the Last 10 Years, for the Specialty AND Causality Proportion
      Emergency Medicine 29,571 9377 31.7%
      p < 0.0001, χ2 test between Emergency Medicine and other specialties searched.
      Cardiology 71,575 28,773 40.2%
      Medical Oncology 42,879 17,614 41.1%
      Neurology 90,277 44,017 48.8%
      Endocrinology 44,596 22,475 50.4%
      Gastroenterology 48,305 24,959 51.7%
      Dermatology 47,905 25,011 52.2%
      Pulmonary medicine 47,470 25,201 53.1%
      Nephrology 31,226 17,226 55.2%
      Rheumatology 23,059 12,733 55.2%
      Hematology 46,346 26,447 57.1%
      TOTAL (emergency medicine excluded) 493,638 244,456 49.5%
      (Performed April 11, 2009).
      low asterisk p < 0.0001, χ2 test between Emergency Medicine and other specialties searched.
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      References

        • Swap C.J.
        • Nagurney J.T.
        Value and limitations of chest pain history in the evaluation of patients with suspected acute coronary syndromes.
        JAMA. 2005; 294: 2623-2629
        • Chagnon I.
        • Bounameaux H.
        • Aujesky D.
        • et al.
        Comparison of two clinical prediction rules and implicit assessment among patients with suspected pulmonary embolism.
        Am J Med. 2002; 113: 269-275
        • Sykes A.
        • Johnston S.L.
        Etiology of asthma exacerbations.
        J Allergy Clin Immunol. 2008; 122: 685-688